Spinal cord infarctions are far less common than cerebral infarcts. Symptoms may include sudden, severe back pain and the acute onset of neurologic deficits over several minutes. Neurologic deficits depend on the level of spinal cord involvement and can include motor weakness, sensory disturbances, and bowel and bladder dysfunction. The vast majority of spinal cord infarctions occur in the territory of the anterior spinal artery, which supplies blood flow to the areas controlling pain, temperature, and touch. The posterior spinal artery is rarely involved, and supplies the areas that control proprioception and vibration sensation. A few causes of anterior spinal artery occlusion include aortic dissection, atherosclerotic embolization, trauma, AVMs, and external compression from a herniated disk, tumor, or fracture. Additionally, arteritis caused by lupus, syphilis, or other conditions may compromise blood flow to the anterior spinal artery. Diabetes, hypertension, and hyperlipidemia all increase the risk of spinal cord infarction. The treatment of spinal cord infarctions will depend on the underlying cause.