RT Book, Section
A1 Burton, John H.
A1 Fortuna, Timothy J.
A2 Tintinalli, Judith E.
A2 Ma, O. John
A2 Yealy, Donald M.
A2 Meckler, Garth D.
A2 Stapczynski, J. Stephan
A2 Cline, David M.
A2 Thomas, Stephen H.
SR Print(0)
ID 1167030944
T1 Joints and Bursae
T2 Tintinalli's Emergency Medicine: A Comprehensive Study Guide, 9e
YR 2020
FD 2020
PB McGraw-Hill Education
PP New York, NY
SN 9781260019933
LK accessemergencymedicine.mhmedical.com/content.aspx?aid=1167030944
RD 2024/04/20
AB Many  mechanisms  provoke  acute  joint  symptoms:  degradation  and  degeneration  of  articular  cartilage  (osteoarthritis),  deposition  of  immune  complexes  or  immune  system–related  phenomena  (rheumatoid  arthritis,  rheumatic  fever,  and  possibly,  a  component  of  gonococcal  arthritis),  crystal-induced  inflammation  (gout  and  pseudogout),  seronegative  spondyloarthropathies  (ankylosing  spondylitis  [see  Chapter  282,  “Systemic  Rheumatic  Diseases”]  and  reactive  arthritis  [postinfectious  with  HLA-B27  susceptibility]),  and  bacterial  or  viral  invasion  (gonococcal  and  nongonococcal  septic  arthritis,  including  Lyme  arthritis).  These  processes  impact  joint  capsules  and  surfaces,  resulting  in  a  cascade  of  reactive  and  inflammatory  events.  This  chapter  reviews  the  common  causes  and  treatments  of  acute  nontraumatic  joint  pain.  Joint  injuries  are  discussed  in  Section  22,  “Orthopedics,”  and  disorders  due  to  repetitive  use  syndromes  are  discussed  throughout  Section  23,  “Musculoskeletal  Disorders,”  by  anatomic  site.