++
Stasis dermatitis results from venous insufficiency, is characteristically distributed on the distal tibia above the medial malleolus, and appears early with erythematous patches. These can progress to scaling as well as eczematous and weeping plaques. Patients will often have light brown pigmentation distributed on the lower third of the extremity due to microvasculature blood extravasation (hemosiderin deposition secondary to increased superficial capillary pressure). Varicose veins are usually present, although they are often difficult to visualize in obese patients. Patients with heart failure, cirrhosis, nephrotic syndrome, or lower extremity trauma are at increased risk due to a chronic edematous state.
+++
Management and Disposition
++
Referral to a primary care physician should be initiated to address the underlying etiology (eg, venous valvular insufficiency, thromboembolic disease, chronic edematous state), and dermatology referral should be made to ensure other diagnoses are not playing a role. Daily elevation and compression hose use should be encouraged. Emollients and mid-potency topical steroids help decrease the pruritus and promote healing.
++
++
Differentiation of stasis dermatitis and early cellulitis can be difficult. Clinical history and deviation from previous presentations may be helpful. Close follow-up for reevaluation should be obtained.
Due to the chronic and repetitive nature of stasis dermatitis, patients use many over-the-counter topical products. These may contribute to allergic and irritant contact dermatitis.
An associated contact dermatitis may initiate an “autosensitization” rash, erythematous patches on the legs and arms. Stop nonessential topical medications or other products.
++