RT Book, Section
A1 Kelen, Gabor D.
A1 Nicolaou, David D.
A1 Cline, David M.
A2 Tintinalli, Judith E.
A2 Stapczynski, J. Stephan
A2 Ma, O. John
A2 Yealy, Donald M.
A2 Meckler, Garth D.
A2 Cline, David M.
SR Print(0)
ID 1153279612
T1 Acid-Base Disorders
T2 Tintinalli’s Emergency Medicine: A Comprehensive Study Guide, 8e
YR 2016
FD 2016
PB McGraw-Hill Education
PP New York, NY
SN 9780071794763
LK accessemergencymedicine.mhmedical.com/content.aspx?aid=1153279612
RD 2024/04/18
AB Controversy  has  existed  concerning  acid-base  physiology  and  the  ideal  method  to  assess  acid-base  disorders  for  130  years.1  The  two  most  common  methods  advocated  to  analyze  acid-base  disorders  are  the  traditional  bicarbonate-centered  method2,3  and  the  Stewart,  or  strong  ion,  method.4,5  The  traditional  approach  teaches  that  acid-base  homeostasis  is  maintained  by  respiratory  control  of  the  partial  pressure  of  carbon  dioxide  (Pco2)  through  changes  in  alveolar  ventilation  and  control  of  HCO3–  reabsorption  and  H+  excretion  by  the  kidneys.  Peter  Stewart  proposed  that  acid-base  physiology  involves  the  dynamic  interaction  of  body  fluids  and  multiple  chemical  species  including  strong  ions  (primarily  Na+,  K+,  Ca2+,  Mg2+,  and  Cl–)  and  weak  acids,  as  well  as  Pco2  control  by  the  lungs.